Prof. Dr. Dunja Bruder

• 1990 – 1996
  Studied biotechnology at Braunschweig University of Technology, Germany
  
  

• 1996
  Basel Institute for Immunology, Switzerland
  
  

• 1996 – 1999
  Doctoral studies at the Helmholtz Centre for Infection Research (HZI), Germany
  
  

• 1999 – 2006
  Research associate at the Helmholtz Centre for Infection Research, in the Mucosal Immunity junior research group
  
  

• 1999
  Hospital Necker, Paris, France
  
  

• 2002
  Dana Farber Cancer Institute, Harvard Medical School, Boston, MA, USA
  
  

• 2003
  Dana Farber Cancer Institute, Harvard Medical School, Boston, MA, USA
  
  

• 2004 – 2005
  Yale University School of Medicine, Division of Pulmonary and Critical Care Medicine, New Haven, CT, USA
  
  

• Since 2006
  Head of the research group Immune Regulation, Helmholtz Centre for Infection Research
  
  

• 2009
  Habilitation (postdoctoral qualification) in Immunology at Hannover Medical School, Germany
  
  

• 2011
  Since August: W2 professor of immunology, University of Magdeburg, Germany, and concurrently head of the research group Immune Regulation, Helmholtz Centre for Infection Research
  
  

• Bruder, D. et al.: Neuropilin-1 – a surface marker of regulatory T-cells. In: Eur. J. Immunol. 34, 2004. S. 623-630.

• Bruder, D. et al.: CD4 T lymphocyte-mediated lung disease: steady state between pathological and tolerogenic immune reactions. In: Am. J. Respir. Crit. Care Med. 170, 2004. S. 1145-1152.

• Westendorf, A.M. et al.: CD4+ T cell mediated intestinal immunity: chronic inflammation versus immune regulation. In: Gut 54, 2005. S. 60-69.

• Bruder, D. et al.: On the edge of autoimmunity: T cell stimulation by steady state dendritic cells prevents autoimmune diabetes. In: Diabetes 54, 2005. S. 3395-3401.

• Bruder, D. et al.: Multiple synergizing factors contribute to the strength of the CD8+ T cell response against listeriolysin O. In: International Immunology 18(1), 2006. S. 89-100.

• Westendorf, A.M. et al.: Autoimmune-mediated intestinal inflammation – impact and regulation of antigen specific CD8+ T cells. In: Gastroenterology 131(2), 2006. S. 510-524.

• Gereke, M. et al.: Phenotypic alterations in type II alveolar epithelial cells in CD4+ T cell mediated lung inflammation. In: Respiratory Research 8, 2007. S. 47.

• Hansen, W. et al.: Chronic antigen stimulation in vivo induces a distinct population of antigen-specific Foxp3lowCD25-regulatory T cells. In: Journal of Immunology 179, 2007. S. 8059-8068.

• Gereke, M. et al.: Type II alveolar epithelial cells present antigen to CD4+ T cells and induce Foxp3+ regulatory T cells. In: Am. J. Resp. Crit. Care Med. 179(5), 2009. S. 344-355.

• Stegemann, S. et al.: Increased susceptibility for superinfection with Streptococcus pneumoniae during influenza virus infection is not caused by TLR7-mediated lymphopenia. In: PLoS ONE 4(3), 2009. e4840.

• Bar-On, L. et al.: CX3CR1pos CD8?pos dendritic cells are a steady state population related to plasmacytoid dendritic cells. In: PNAS 2010 (Epub ahead of print).

• Project leader in Collaborative Research Centres SFB 587 and 738 (also in SFB 621 until 06/2009); project leader in the Helmholtz Alliance on Immunotherapy of Cancer

• Member of the German Society for Immunology

• Member of the German Association of University Professors and Lecturers

• Member at the Infection Biology Centre (ZIB), Hannover Medical School (MHH), Germany

• Member of the MD/PhD programme Molecular Medicine, MHH

• Member of independent junior research group (IRTG 1273), MHH

• Member of the Helmholtz Centre for Infection Research (HZI) Graduate School

• Ad hoc referee for various international journals

• Participated in the Helmholtz Management Academy, the optimal preparation for future management positions, 2009 – 2011

Additional interests

• Experiencing nature

Soft Skills

• Trained as an internal mediator

• In addition to carrying out first-class research, I consider the following to be particularly important: supporting the young researchers under me, teaching well, and having an interpersonal relationship based on trust with the members of my research group.

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