Short CV/Education and training

  • 1990 – 1996
    Studied biotechnology at Braunschweig University of Technology, Germany

  • 1996
    Basel Institute for Immunology, Switzerland

  • 1996 – 1999
    Doctoral studies at the Helmholtz Centre for Infection Research (HZI), Germany

  • 1999 – 2006
    Research associate at the Helmholtz Centre for Infection Research, in the Mucosal Immunity junior research group

  • 1999
    Hospital Necker, Paris, France

  • 2002
    Dana Farber Cancer Institute, Harvard Medical School, Boston, MA, USA

  • 2003
    Dana Farber Cancer Institute, Harvard Medical School, Boston, MA, USA

  • 2004 – 2005
    Yale University School of Medicine, Division of Pulmonary and Critical Care Medicine, New Haven, CT, USA

  • Since 2006
    Head of the research group Immune Regulation, Helmholtz Centre for Infection Research

  • 2009
    Habilitation (postdoctoral qualification) in Immunology at Hannover Medical School, Germany

  • 2011
    Since August: W2 professor of immunology, University of Magdeburg, Germany, and concurrently head of the research group Immune Regulation, Helmholtz Centre for Infection Research

Selected publications

  • Bruder, D. et al.: Neuropilin-1 – a surface marker of regulatory T-cells. In: Eur. J. Immunol. 34, 2004. S. 623-630.

  • Bruder, D. et al.: CD4 T lymphocyte-mediated lung disease: steady state between pathological and tolerogenic immune reactions. In: Am. J. Respir. Crit. Care Med. 170, 2004. S. 1145-1152.

  • Westendorf, A.M. et al.: CD4+ T cell mediated intestinal immunity: chronic inflammation versus immune regulation. In: Gut 54, 2005. S. 60-69.

  • Bruder, D. et al.: On the edge of autoimmunity: T cell stimulation by steady state dendritic cells prevents autoimmune diabetes. In: Diabetes 54, 2005. S. 3395-3401.

  • Bruder, D. et al.: Multiple synergizing factors contribute to the strength of the CD8+ T cell response against listeriolysin O. In: International Immunology 18(1), 2006. S. 89-100.

  • Westendorf, A.M. et al.: Autoimmune-mediated intestinal inflammation – impact and regulation of antigen specific CD8+ T cells. In: Gastroenterology 131(2), 2006. S. 510-524.

  • Gereke, M. et al.: Phenotypic alterations in type II alveolar epithelial cells in CD4+ T cell mediated lung inflammation. In: Respiratory Research 8, 2007. S. 47.

  • Hansen, W. et al.: Chronic antigen stimulation in vivo induces a distinct population of antigen-specific Foxp3lowCD25-regulatory T cells. In: Journal of Immunology 179, 2007. S. 8059-8068.

  • Gereke, M. et al.: Type II alveolar epithelial cells present antigen to CD4+ T cells and induce Foxp3+ regulatory T cells. In: Am. J. Resp. Crit. Care Med. 179(5), 2009. S. 344-355.

  • Stegemann, S. et al.: Increased susceptibility for superinfection with Streptococcus pneumoniae during influenza virus infection is not caused by TLR7-mediated lymphopenia. In: PLoS ONE 4(3), 2009. e4840.

  • Bar-On, L. et al.: CX3CR1pos CD8?pos dendritic cells are a steady state population related to plasmacytoid dendritic cells. In: PNAS 2010 (Epub ahead of print).

Selected projects

  • Project leader in Collaborative Research Centres SFB 587 and 738 (also in SFB 621 until 06/2009); project leader in the Helmholtz Alliance on Immunotherapy of Cancer

Membership in scientific bodies/juries

  • Member of the German Society for Immunology

  • Member of the German Association of University Professors and Lecturers

  • Member at the Infection Biology Centre (ZIB), Hannover Medical School (MHH), Germany

  • Member of the MD/PhD programme Molecular Medicine, MHH

  • Member of independent junior research group (IRTG 1273), MHH

  • Member of the Helmholtz Centre for Infection Research (HZI) Graduate School

  • Ad hoc referee for various international journals

Additional qualifications

  • Participated in the Helmholtz Management Academy, the optimal preparation for future management positions, 2009 – 2011

Soft Skills/Other activities and achievements

Soft Skills

  • Trained as an internal mediator

  • In addition to carrying out first-class research, I consider the following to be particularly important: supporting the young researchers under me, teaching well, and having an interpersonal relationship based on trust with the members of my research group.


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  1. Too Much of a Good Thing

    Our immune system fights pathogens, but it sometimes overshoots the mark and damages body tissue. Researchers from the Helmholtz Centre for Infection Research HZI report that activation of a molecule called ICOS, on the surface of immune cells, limits the extent of lung damage during the infection.

  2. Appetite Suppressant for Scavenger Cells

    When infected with influenza, the body becomes an easy target for bacteria. Now, a team of immunologists at the Helmholtz Centre for Infection Research HZI and partners has discovered how the flu virus alters the host's immune system and compromises its capacity to effectively fight off bacterial infections.

  3. The Mystery of the Third Signal

    In healthy lungs, several mechanisms prevent the body's immune system to attack lung tissue. But in case of chronic obstructive pulmonary disease (COPD), these mechanisms cease to function properly. The research group of Prof. Dunja Bruder at the Helmholtz Centre for Infection Research (HZI) in Braunschweig, Germany, has explored and described these processes in great detail.

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